Former Clinical PhD student awarded prestigious Wellcome Fellowship
Kashyap Patel, a former clinical PhD student in Kei Sakamoto's lab in the MRC PPU, has been awarded a highly competitive Wellcome Trust Clinical Postdoctoral Fellowship to undertake research at the University of Exeter into the genetic basis of Diabetes.
The Wellcome Trust Postdoctoral Fellowship scheme was established to support the most talented clinicians wishing to pursue a scientific career path towards independence.
During his PhD studies, Kash discovered a critical role for the salt-inducible kinase (SIK1, 2 and 3) as suppressors of gluconeogenesis in the liver that was subsequently published in Nature Communications.
He then moved to the University of Exeter where he was appointed Clinical Lecturer in the world-leading laboratory of Andrew Hattersley FRS and completed his clinical training in Diabetes and Endocrinology. Over the last 18 months he has developed genetic methodologies that have transformed understanding of diabetes occurring before the age of 6 months. With support from the Wellcome Fellowship, Kash will now build on this advance to identify novel genes that cause Mendelian-inherited forms of Diabetes.
The Wellcome Trust Postdoctoral Fellowship scheme was established to support the most talented clinicians wishing to pursue a scientific career path towards independence.
During his PhD studies, Kash discovered a critical role for the salt-inducible kinase (SIK1, 2 and 3) as suppressors of gluconeogenesis in the liver that was subsequently published in Nature Communications.
He then moved to the University of Exeter where he was appointed Clinical Lecturer in the world-leading laboratory of Andrew Hattersley FRS and completed his clinical training in Diabetes and Endocrinology. Over the last 18 months he has developed genetic methodologies that have transformed understanding of diabetes occurring before the age of 6 months. With support from the Wellcome Fellowship, Kash will now build on this advance to identify novel genes that cause Mendelian-inherited forms of Diabetes.